Logo: to the web site of Uppsala University

uu.sePublikasjoner fra Uppsala universitet
Endre søk
RefereraExporteraLink to record
Permanent link

Direct link
Referera
Referensformat
  • apa
  • ieee
  • modern-language-association
  • vancouver
  • Annet format
Fler format
Språk
  • de-DE
  • en-GB
  • en-US
  • fi-FI
  • nn-NO
  • nn-NB
  • sv-SE
  • Annet språk
Fler språk
Utmatningsformat
  • html
  • text
  • asciidoc
  • rtf
TGFß/Smad signaling in epithelial to mesenchymal transition
Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska och farmaceutiska vetenskapsområdet, centrumbildningar mm, Ludwiginstitutet för cancerforskning.
Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska och farmaceutiska vetenskapsområdet, centrumbildningar mm, Ludwiginstitutet för cancerforskning.
Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska och farmaceutiska vetenskapsområdet, centrumbildningar mm, Ludwiginstitutet för cancerforskning.
2006 (engelsk)Inngår i: Smad Signal Transduction: Smads in Proliferation, Differentiation and Disease / [ed] P. ten Dijke and C.-H. Heldin, Dordrecht: Springer , 2006, s. 131-150Kapittel i bok, del av antologi (Fagfellevurdert)
Abstract [en]

Epithelial-mesenchymal transition (EMT) allows polarized epithelial layers to locally dissolve and create fibroblast-like or myofibroblastic cell derivatives. EMT resembles other processes of transdifferentiation from one differentiated cell type to another and is critical for proper embryonic development. Furthermore, EMT describes similar processes between endothelial and myoepithelial cells occurring during blood vessel remodeling. All such processes are triggered and controlled by the concerted action of multiple extracellular growth/morphogenetic factors, TGF-β being one of them. The mechanism by which TGF-β elicits EMT has been a major topic of current research. This is due to the relevance EMT has to the processes of tumor cell invasiveness and metastasis on the one hand, and to fibrotic conditions on the other. Here we emphasize on mechanisms of signal transduction and specific gene targets of the TGF-β pathway that are critical effectors of EMT.

sted, utgiver, år, opplag, sider
Dordrecht: Springer , 2006. s. 131-150
Serie
Proteins and Cell Regulation ; 5
Emneord [en]
Epithelial–mesenchymal transition, metastasis, Smad, signal transduction, TGFbeta, tumour suppression
HSV kategori
Identifikatorer
URN: urn:nbn:se:uu:diva-11616ISBN: 978-1-4020-4709-1 (tryckt)OAI: oai:DiVA.org:uu-11616DiVA, id: diva2:39385
Tilgjengelig fra: 2007-10-05 Laget: 2007-10-05 Sist oppdatert: 2018-06-04bibliografisk kontrollert

Open Access i DiVA

Fulltekst mangler i DiVA

Person

Moustakas, AristidisThuault, Sylvie

Søk i DiVA

Av forfatter/redaktør
Moustakas, AristidisThuault, Sylvie
Av organisasjonen

Søk utenfor DiVA

GoogleGoogle Scholar

isbn
urn-nbn

Altmetric

isbn
urn-nbn
Totalt: 394 treff
RefereraExporteraLink to record
Permanent link

Direct link
Referera
Referensformat
  • apa
  • ieee
  • modern-language-association
  • vancouver
  • Annet format
Fler format
Språk
  • de-DE
  • en-GB
  • en-US
  • fi-FI
  • nn-NO
  • nn-NB
  • sv-SE
  • Annet språk
Fler språk
Utmatningsformat
  • html
  • text
  • asciidoc
  • rtf