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Cholecystokinin-Like Peptide (DSK) in Drosophila, Not Only for Satiety Signaling
Uppsala University, Disciplinary Domain of Medicine and Pharmacy, Faculty of Medicine, Department of Neuroscience, Functional Pharmacology.
2014 (English)In: Frontiers in Endocrinology, ISSN 1664-2392, E-ISSN 1664-2392, Vol. 5, 219- p.Article in journal (Refereed) Published
Abstract [en]

Cholecystokinin (CCK) signaling appears well conserved over evolution. In Drosophila, the CCK-like sulfakinins (DSKs) regulate aspects of gut function, satiety and food ingestion, hyperactivity and aggression, as well as escape-related locomotion and synaptic plasticity during neuromuscular junction development. Activity in the DSK-producing neurons is regulated by octopamine. We discuss mechanisms behind CCK function in satiety, aggression, and locomotion in some detail and highlight similarities to mammalian CCK signaling.

Place, publisher, year, edition, pages
2014. Vol. 5, 219- p.
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Biological Sciences
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URN: urn:nbn:se:uu:diva-241910DOI: 10.3389/fendo.2014.00219PubMedID: 25566191OAI: oai:DiVA.org:uu-241910DiVA: diva2:781877
Available from: 2015-01-19 Created: 2015-01-19 Last updated: 2017-12-05Bibliographically approved

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Williams, Michael J

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