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Neurofunctional deficits and potentiated apoptosis by neonatal NMDA antagonist administration.
Uppsala University, Medicinska vetenskapsområdet, Faculty of Medicine, Department of Neuroscience. Teknisk-naturvetenskapliga vetenskapsområdet, Faculty of Science and Technology, Biology, Department of Physiology and Developmental Biology, Environmental Toxicology. (Enh för psykiatri Ulleråker)
Teknisk-naturvetenskapliga vetenskapsområdet, Faculty of Science and Technology, Biology, Department of Physiology and Developmental Biology, Environmental Toxicology.
Department of Surgical Sciences. Teknisk-naturvetenskapliga vetenskapsområdet, Faculty of Science and Technology, Biology, Department of Physiology and Developmental Biology, Environmental Toxicology.
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2004 (English)In: Behav Brain Res, ISSN 0166-4328, Vol. 153, no 2, 367-76 p.Article in journal (Refereed) Published
Place, publisher, year, edition, pages
2004. Vol. 153, no 2, 367-76 p.
Keyword [en]
Animals, Animals; Newborn, Apoptosis/*drug effects, Brain/*drug effects/growth & development, Comparative Study, Diazepam/pharmacology, Drug Interactions, Female, Injections; Subcutaneous, Ketamine/*toxicity, Male, Maze Learning/*drug effects, Mice, Motor Activity/*drug effects, Neurons/drug effects, Parietal Lobe/drug effects/growth & development, Posterior Thalamic Nuclei/drug effects/growth & development, Pregnancy, Receptors; GABA-A/agonists, Receptors; N-Methyl-D-Aspartate/*antagonists & inhibitors
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URN: urn:nbn:se:uu:diva-70798PubMedID: 15265631OAI: oai:DiVA.org:uu-70798DiVA: diva2:98709
Available from: 2006-03-20 Created: 2006-03-20 Last updated: 2011-01-12

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Fredriksson, AndersAlm, HenrikGordh, TorstenEriksson, Per

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