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  • 51.
    Sollie, Ove
    et al.
    Norwegian Sch Sports Sci, Dept Phys Performance, Sognsveien 220,POB 4014 Ulleval Stad, N-0806 Oslo, Norway.
    Jeppesen, Per Bendix
    Aarhus Univ, Dept Clin Med, Aarhus, Denmark.
    Tangen, Daniel Steensen
    Norwegian Sch Sports Sci, Dept Phys Performance, Sognsveien 220,POB 4014 Ulleval Stad, N-0806 Oslo, Norway.
    Jernerén, Fredrik
    University of Oxford, Department of Pharmacology.
    Nellemann, Birgitte
    Norwegian Sch Sports Sci, Dept Phys Performance, Sognsveien 220,POB 4014 Ulleval Stad, N-0806 Oslo, Norway.
    Valsdottir, Ditta
    Norwegian Sch Sports Sci, Dept Phys Performance, Sognsveien 220,POB 4014 Ulleval Stad, N-0806 Oslo, Norway; Atlantis Med Univ Coll, Dept Med Sci, Oslo, Norway.
    Madsen, Klavs
    Norwegian Sch Sports Sci, Dept Phys Performance, Sognsveien 220,POB 4014 Ulleval Stad, N-0806 Oslo, Norway; Aarhus Univ, Dept Publ Hlth Sport Sci, Aarhus, Denmark.
    Turner, Cheryl
    Univ Oxford, Dept Pharmacol, Oxford, England.
    Refsum, Helga
    Univ Oxford, Dept Pharmacol, Oxford, England; Univ Oslo, Dept Nutr, Sect Mol Nutr, Oslo, Norway.
    Skålhegg, Bjørn Steen
    Univ Oslo, Dept Nutr, Sect Mol Nutr, Oslo, Norway.
    Ivy, John L
    Univ Texas Austin, Dept Kinesiol & Hlth Educ, Austin, TX 78712 USA.
    Jensen, Jørgen
    Norwegian Sch Sports Sci, Dept Phys Performance, Sognsveien 220,POB 4014 Ulleval Stad, N-0806 Oslo, Norway.
    Protein intake in the early recovery period after exhaustive exercise improves performance the following day.2018Inngår i: Journal of applied physiology, ISSN 8750-7587, E-ISSN 1522-1601, Vol. 125, nr 6, s. 1731-1742Artikkel i tidsskrift (Fagfellevurdert)
    Abstract [en]

    The aim of the present study was to investigate the effect of protein and carbohydrate ingestion during early recovery from exhaustive exercise on performance after 18 h recovery. Eight elite cyclists (VO2max 74.0±1.6 ml∙kg-1∙min-1) completed two exercise and diet interventions in a double-blinded, randomized, crossover design. Participants cycled first at 73% of VO2max (W73%) followed by one-min intervals at 90% of VO2max until exhaustion. During the first two hours of recovery, participants ingested either 1.2 g carbohydrate∙kg-1∙h-1 (CHO) or 0.8 g carbohydrate + 0.4 g protein∙kg-1∙h-1 (CHO+PROT). The diet during the remaining recovery period was similar for both interventions and adjusted to body weight. After an 18 h recovery, cycling performance was assessed with a 10 s sprint test, 30 min of cycling at W73%, and a cycling time trial (TT). The TT was 8.5% faster (41:53±1:51 min vs 45:26±1:32 min; p<0.03) after CHO+PROT compared to CHO. Mean power output during the sprints was 3.7% higher in CHO-PROT compared to CHO (1063±54 W vs 1026±53 W; p<0.01). Nitrogen balance in the recovery period was negative in CHO and neutral in CHO+PROT (-82.4±11.5 vs 7.0±15.4 mg∙kg-1; p<0.01).

    IN CONCLUSION: TT and sprint performances were improved 18 h after exhaustive cycling by CHO-PROT supplementation during the first two hours of recovery compared with isoenergetic CHO supplementation. Our results indicate that intake of carbohydrate plus protein after exhaustive endurance exercise more rapidly converts the body from a catabolic to an anabolic state than carbohydrate alone, thus speeding recovery and improving subsequent cycling performance.

  • 52.
    Suijkerbuijk, Mathijs A. M.
    et al.
    Univ Med Ctr Rotterdam, Dept Orthopaed Surg, Erasmus MC, Rotterdam, Netherlands.
    Ponzetti, Marco
    Univ Aquila, Dept Biotechnol & Appl Clin Sci, Laquila, Italy.
    Rahim, Masouda
    Univ Cape Town, Fac Hlth Sci, Dept Human Biol, Div Exercise Sci & Sports Med, Cape Town, South Africa.
    Posthumus, Michael
    Univ Cape Town, Fac Hlth Sci, Dept Human Biol, Div Exercise Sci & Sports Med, Cape Town, South Africa.
    Häger, Charlotte K.
    Umea Univ, Dept Community Med & Rehabil, Umea, Sweden.
    Stattin, Evalena
    Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska fakulteten, Institutionen för immunologi, genetik och patologi, Medicinsk genetik och genomik. Uppsala universitet, Science for Life Laboratory, SciLifeLab.
    Nilsson, Kjell G.
    Umea Univ, Dept Surg & Perioperat Sci, Umea, Sweden.
    Teti, Anna
    Univ Aquila, Dept Biotechnol & Appl Clin Sci, Laquila, Italy.
    Meuffels, Duncan E.
    Univ Med Ctr Rotterdam, Dept Orthopaed Surg, Erasmus MC, Rotterdam, Netherlands.
    van der Eerden, Bram J. C.
    Univ Med Ctr Rotterdam, Dept Internal Med, Erasmus MC, Rotterdam, Netherlands.
    Collins, Malcolm
    Univ Cape Town, Fac Hlth Sci, Dept Human Biol, Div Exercise Sci & Sports Med, Cape Town, South Africa.
    September, Alison, V
    Univ Cape Town, Fac Hlth Sci, Dept Human Biol, Div Exercise Sci & Sports Med, Cape Town, South Africa.
    Functional polymorphisms within the inflammatory pathway regulate expression of extracellular matrix components in a genetic risk dependent model for anterior cruciate ligament injuries2019Inngår i: Journal of Science and Medicine in Sport, ISSN 1440-2440, E-ISSN 1878-1861, Vol. 22, nr 11, s. 1219-1225Artikkel i tidsskrift (Fagfellevurdert)
    Abstract [en]

    Objectives: To investigate the functional effect of genetic polymorphisms of the inflammatory pathway on structural extracellular matrix components (ECM) and the susceptibility to an anterior cruciate ligament (ACL) injury. Design: Laboratory study, case-control study. Methods: Eight healthy participants were genotyped for interleukin (IL)1B rs16944 C> Tand IL6 rs1800795 G >C and classified into genetic risk profile groups. Differences in type I collagen (COL1A1), type V collagen (COL5A1), biglycan (BGN) and decorin (DCN) gene expression were measured in fibroblasts either unstimulated or following IL-1 beta, IL-6 or tumor necrosis factor (TNF)-alpha treatment. Moreover, a genetic association study was conducted in: (i) a Swedish cohort comprised of 116 asymptomatic controls (CON) and 79 ACL ruptures and (ii) a South African cohort of 100 CONs and 98 ACLs. Participants were genotyped for COL5A1 rs12722 C >T, IL1B rs16944 C >T, IL6 rs1800795 G> C and IL6R rs2228145 G >C. Results: IL1B high-risk fibroblasts had decreased BGN (p = 0.020) and COL5A1 (p = 0.012) levels after IL-1 beta stimulation and expressed less COL5A1 (p = 0.042) following TNF-alpha treatment. Similarly, unstimulated IL6 high-risk fibroblasts had lower COL5A1 (p = 0.012) levels than IL6 low-risk fibroblasts. In the genetic association study, the COL5A1-IL1B-IL6 T-C-G (p = 0.034, Haplo-score 2.1) and the COL5A1-IL1B-1L6R T-C-A (p = 0.044, Haplo-score: 2.0) combinations were associated with an increased susceptibility to ACL injury in the Swedish cohort when only male participants were evaluated. Conclusions: This study shows that polymorphisms within genes of the inflammatory pathway modulate the expression of structural and fibril-associated ECM components in a genetic risk depended manner, contributing to an increased susceptibility to ACL injuries. (C) 2019 Sports Medicine Australia. Published by Elsevier Ltd. All rights reserved.

  • 53.
    Svensson, Martina
    et al.
    Lund Univ, Dept Expt Med Sci, Expt Neuroinfiammat Lab, BMC B11, S-22184 Lund, Sweden.
    Brundin, Lena
    Van Andel Res Inst, Ctr Neurodegenerat Sci, Grand Rapids, MI USA.
    Erhardt, Sophie
    Karolinska Inst, Dept Physiol & Pharmacol, Stockholm, Sweden.
    Madaj, Zachary
    Van Andel Res Inst, Ctr Neurodegenerat Sci, Grand Rapids, MI USA.
    Hållmarker, Ulf
    Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska fakulteten, Institutionen för medicinska vetenskaper, Kardiologi. Mora Hosp, Dept Internal Med, Mora, Sweden.
    James, Stefan
    Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska fakulteten, Institutionen för medicinska vetenskaper. Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska och farmaceutiska vetenskapsområdet, centrumbildningar mm, Uppsala kliniska forskningscentrum (UCR).
    Deierborg, Tomas
    Lund Univ, Dept Expt Med Sci, Expt Neuroinfiammat Lab, BMC B11, S-22184 Lund, Sweden.
    Long distance ski racing is associated with lower long-term incidence of depression in a population based, large-scale study2019Inngår i: Psychiatry Research, ISSN 0165-1781, E-ISSN 1872-7123, Vol. 281, artikkel-id 112546Artikkel i tidsskrift (Fagfellevurdert)
    Abstract [en]

    Physical activity has been proposed to be beneficial for prevention of depression, although the importance of exercise intensity, sex-specific mechanisms, and duration of the effects need to be clarified. Using an observational study design, following 395,369 individuals up to 21 years we studied whether participation in an ultralong-distance cross-country ski race was associated with lower risk of developing depression. Skiers (participants in the race) and matched non-skiers from the general population (non-participants in the race) were studied after participation (same year for non-participation) in the race using the Swedish population and patient registries. The risk of depression in skiers (n = 197,685, median age 36 years, 38% women) was significantly lower, to nearly half of that in non-skiers (adjusted hazard ratio, HR 0.53) over the follow-up period. Further, a higher fitness level (measured as the finishing time to complete the race, a proxy for higher exercise dose) was associated with lower incidence of depression in men (adjusted HR 0.65), but not in women. Our results support the recommendations of engaging in physical activity as a preventive strategy decreasing the risk for depression in both men and women. Furthermore, the exercise could reduce risk for depression in a dose-dependent matter, in particular in males.

  • 54.
    Timpka, Toomas
    et al.
    Linkoping Univ, Athlet Res Ctr, Linkoping, Sweden;Linkoping Univ, Dept Med & Hlth Sci, SE-58183 Linkoping, Sweden;Reg Ostergotland, Ctr Healthcare Dev, Unit Hlth Anal, Linkoping, Sweden.
    Janson, Staffan
    Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska fakulteten, Institutionen för kvinnors och barns hälsa.
    Jacobsson, Jenny
    Linkoping Univ, Athlet Res Ctr, Linkoping, Sweden;Linkoping Univ, Dept Med & Hlth Sci, SE-58183 Linkoping, Sweden.
    Dahlstrom, Orjan
    Linkoping Univ, Athlet Res Ctr, Linkoping, Sweden;Linkoping Univ, Dept Behav Sci & Learning, Linkoping, Sweden.
    Spreco, Armin
    Linkoping Univ, Athlet Res Ctr, Linkoping, Sweden;Linkoping Univ, Dept Med & Hlth Sci, SE-58183 Linkoping, Sweden;Reg Ostergotland, Ctr Healthcare Dev, Unit Hlth Anal, Linkoping, Sweden.
    Kowalski, Jan
    Linkoping Univ, Athlet Res Ctr, Linkoping, Sweden;Linkoping Univ, Dept Med & Hlth Sci, SE-58183 Linkoping, Sweden.
    Bargoria, Victor
    Linkoping Univ, Athlet Res Ctr, Linkoping, Sweden;Linkoping Univ, Dept Med & Hlth Sci, SE-58183 Linkoping, Sweden;Moi Univ, Dept Orthopaed & Rehabil, Eldoret, Kenya.
    Mountjoy, Margo
    McMaster Univ, Dept Family Med, Hamilton, ON, Canada;Int Olymp Comm, Med Commiss, Games Grp, Lausanne, Switzerland.
    Svedin, Carl Goran
    Linkoping Univ, Athlet Res Ctr, Linkoping, Sweden;Linkoping Univ, Dept Clin & Expt Med, Barnafrid, Linkoping, Sweden.
    Lifetime history of sexual and physical abuse among competitive athletics (track and field) athletes: cross sectional study of associations with sports and non-sports injury2019Inngår i: British Journal of Sports Medicine, ISSN 0306-3674, E-ISSN 1473-0480, Vol. 53, nr 22, s. 1412-+Artikkel i tidsskrift (Fagfellevurdert)
    Abstract [en]

    Objective To examine associations between lifetime sexual and physical abuse, and the likelihood of injury within and outside sport in athletes involved in competitive athletics. Methods A cross sectional study was performed among the top 10 Swedish athletics athletes using 1 year prevalence of sports and non-sports injuries as the primary outcome measure. Associations with sociodemographic characteristics, lifetime abuse history and training load were investigated. Data were analysed using simple and multiple logistic regression models. Results 11% of 197 participating athletes reported lifetime sexual abuse; there was a higher proportion of women (16.2%) than men (4.3%) (P=0.005). 18% reported lifetime physical abuse; there was a higher proportion of men (22.8%) than women (14.3%) (P=0.050). For women, lifetime sexual abuse was associated with an increased likelihood of a non-sports injury (OR 8.78, CI 2.76 to 27.93; P<0.001). Among men, increased likelihood of a non-sports injury was associated with more frequent use of alcoholic beverages (OR 6.47, CI 1.49 to 28.07; P=0.013), while commencing athletics training at >13 years of age was associated with a lower likelihood of non-sports injury (OR 0.09, CI 0.01 to 0.81; P=0.032). Lifetime physical abuse was associated with a higher likelihood of sports injury in women (OR 12.37, CI 1.52 to 100.37; P=0.019). Among men, athletes with each parents with <= 12 years formal education had a lower likelihood of sustaining an injury during their sports practice (OR 0.37, CI 0.14 to 0.96; P=0.040). Conclusions Lifetime sexual and physical abuse were associated with an increased likelihood of injury among female athletes. Emotional factors should be included in the comprehension of injuries sustained by athletes.

  • 55.
    Wesnes, Kristin
    et al.
    Univ Bergen, Dept Clin Med, Bergen, Norway; Haukeland Hosp, Norwegian Multiple Sclerosis Competence Ctr, Dept Neurol, Bergen, Norway.
    Myhr, Kjell-Morten
    Univ Bergen, Dept Clin Med, Bergen, Norway; Haukeland Hosp, Dept Neurol, Norwegian Multiple Sclerosis Registry & Biobank, Bergen, Norway.
    Riise, Trond
    Haukeland Hosp, Norwegian Multiple Sclerosis Competence Ctr, Dept Neurol, Bergen, Norway; Univ Bergen, Dept Global Publ Hlth & Primary Care, Bergen, Norway.
    Cortese, Marianna
    Univ Bergen, Dept Clin Med, Bergen, Norway; Haukeland Hosp, Norwegian Multiple Sclerosis Competence Ctr, Dept Neurol, Bergen, Norway.
    Pugliatti, Maura
    Univ Bergen, Dept Global Publ Hlth & Primary Care, Bergen, Norway; Univ Ferrara, Dept Biomed & Specialty Surg Sci, Sect Clin Neurol, Ferrara, Italy.
    Boström, Inger
    Univ Linköping, Div Neurol, Dept Clin & Expt Med, Linköping, Sweden.
    Landtblom, Anne-Marie
    Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska fakulteten, Institutionen för neurovetenskap, Neurologi. Univ Linköping, Div Neurol, Dept Clin & Expt Med, Linköping, Sweden.
    Wolfson, Christina
    McGill Univ, Dept Epidemiol Biostat & Occupat Hlth, Montreal, PQ, Canada; McGill Univ, Ctr Hlth, Res Inst, Montreal, PQ, Canada.
    Bjørnevik, Kjetil
    Haukeland Hosp, Norwegian Multiple Sclerosis Competence Ctr, Dept Neurol, Bergen, Norway; Univ Bergen, Dept Global Publ Hlth & Primary Care, Bergen, Norway.
    Physical Activity Is Associated With A Decreased Multiple Sclerosis Risk: The EnvIMS study2018Inngår i: Multiple Sclerosis, ISSN 1352-4585, E-ISSN 1477-0970, Vol. 24, nr 2, s. 150-157Artikkel i tidsskrift (Fagfellevurdert)
    Abstract [en]

    Background: The lifestyle factors smoking and obesity have been associated with the risk of multiple sclerosis (MS). Physical activity (PA) may also be of importance.

    Objective: To examine the association between PA and MS risk in Italy, Norway, and Sweden and to evaluate the possible influence by established risk factors.

    Methods: In this case–control study, 1904 cases and 3694 controls were asked to report their average weekly amounts of light and vigorous PA during adolescence on a scale ranging from none to more than 3 hours activity. We used logistic regression to estimate odds ratios (ORs) and 95% confidence intervals (CIs) and adjusted for potential confounders.

    Results: Vigorous PA was inversely associated with MS risk in the pooled analysis (p-trend < 0.001) with an age- and sex-adjusted OR of 0.74 (95% CI: 0.63–0.87) when comparing the highest and lowest levels. Adjusting for outdoor activity, infectious mononucleosis, body size, and smoking yielded similar results. The association was present in all countries and was not affected by exclusion of patients with early disease onset. Light PA was not associated with the risk of MS.

    Conclusion: Our findings suggest that vigorous PA can modify the risk of developing MS independent of established risk factors.

  • 56.
    Westerberg, Elisabet
    et al.
    Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska fakulteten, Institutionen för neurovetenskap, Klinisk neurofysiologi.
    Molin, Carl Johan
    Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska fakulteten, Institutionen för neurovetenskap, Klinisk neurofysiologi.
    Spörndly-Nees, Søren
    Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska fakulteten, Institutionen för neurovetenskap, Fysioterapi.
    Widenfalk, Johan
    Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska fakulteten, Institutionen för neurovetenskap, Klinisk neurofysiologi.
    Rostedt Punga, Anna
    Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, Medicinska fakulteten, Institutionen för neurovetenskap, Klinisk neurofysiologi.
    The impact of physical exercise on neuromuscular function in Myasthenia gravis patients: A single-subject design study2018Inngår i: Medicine (Baltimore, Md.), ISSN 0025-7974, E-ISSN 1536-5964, Vol. 97, nr 31, artikkel-id e11510Artikkel i tidsskrift (Fagfellevurdert)
    Abstract [en]

    There is a need for tailored exercise recommendations to patients with Myasthenia Gravis (MG). A few pilot studies have recently shown that physical exercise in accordance with general recommendations to healthy adults can be applied safely to patients with mild MG symptoms. How physical exercise affects muscle parameters and risk factors for life-style diseases in patients with MG is, however, only poorly known. We evaluated functional skeletal muscle parameters in 11 MG patients, before and after conducting a 12-week supervised physical therapy regimen of aerobic and high-resistance strength training. After the training program, parameters of the proximal leg muscle rectus femoris improved: compound motor action potential (from 4.5 ± 2.6 to 5.3 ± 2.8 mV, p=0.016), isometric muscle force (from 25.2 ± 4.4 to 30.2 ± 3.8 kg; p=0.014) and ultrasound muscle thickness (from 19.6 ± 5.6 to 23.0 ± 3.9 mm, p=0.0098) all increased. Further, physical performance-based measures improved, including the 30-Second Chair Stand Test (median change +2, p=0.0039) as well as the clinical MG composite score (from 3[2-5] to 2 [0-4], p=0.043). These findings indicate that MG patients can improve their functional muscle status as a result of aerobic and high-resistance strength training, especially in proximal leg muscles. This is important knowledge when physical therapy is considered for this patient group, for whom no guidelines on physical exercise currently exist.

  • 57.
    Yngve, Agneta
    et al.
    Unit for Preventive Nutrition, Department of Medical Nutrition, Karolinska Institutet, Stockholm, Sweden.
    Nilsson, Andreas
    Unit for Preventive Nutrition, Department of Medical Nutrition, Karolinska Institutet, Stockholm, Sweden.
    Sjoström, Michael
    Unit for Preventive Nutrition, Department of Medical Nutrition, Karolinska Institutet, Stockholm, Sweden.
    Ekelund, Ulf
    Örebro universitet, Institutionen för hälsovetenskap och medicin.
    Effect of monitor placement and of activity setting on the MTI accelerometer output2003Inngår i: Medicine & Science in Sports & Exercise, ISSN 0195-9131, E-ISSN 1530-0315, Vol. 35, nr 2, s. 320-326Artikkel i tidsskrift (Fagfellevurdert)
    Abstract [en]

    PURPOSE: To examine the effect of monitor placement (hip vs back) and of activity setting (treadmill vs track) on the output from the Manufacturing Technology Inc. (MTI), activity monitor (model WAM 7164).

    METHODS: In a laboratory study, 28 subjects (14 men, 14 women) walked at a normal pace, walked at a fast pace, and jogged at a comfortable pace on an indoor track. These activities were repeated on a treadmill using the individual speeds from the track locomotion. Oxygen uptake was measured simultaneously using a portable metabolic system. One activity monitor was worn on the hip and one on the lower back. In a field study, 34 subjects (18 men, 16 women) each wore two monitors (hip and low back placement) for seven consecutive days. In the laboratory study, ANOVA showed significant effects of placement ( P = 0.009) and setting ( P < 0.001), indicating that activity counts differ between different body sites and different settings (track vs treadmill). Gross energy expenditure predictive equations were developed and thereafter evaluated in the field study. Time spent at moderate and vigorous intensity of physical activity was 38% and 85% ( P < 0.001) higher when calculated from the treadmill-based equations as compared to the track-based equations. Free-living physical activity estimates were not affected by the placement.

    CONCLUSION: The relationship between activity counts and energy expenditure during laboratory locomotion is placement and setting-specific. When habitual physical activity is assessed in free-living subjects, the treadmill derived relationship between energy expenditure and activity counts may overestimate time spent at moderate intensity of physical activity, whereas the placement of the monitor does not influence on the interpretation of the data.

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